Note – This is a personal experiences of the trajectory of my Autism
- Hemiparesis or hemiplegia of the lower half of the contralateral face
- Hemiparesis or hemiplegia of the contralateral upper and lower extremities*
- Sensory loss of the contralateral face, arm and leg*
- Ataxia of contralateral extremities*
- Speech impairments/aphasia: Broca’s, Wernicke’s or Global aphasia as a result of a dominant hemisphere lesion (usually the left brain)
- Perceptual deficits: hemispatial neglect, anosognosia, apraxia, and spatial disorganization as a result of a non-dominant hemisphere lesion (usually the right brain)
- Visual disorders: déviation conjuguée, a gaze preference towards the side of the lesion; contralateral homonymous hemianopsia Overview
Which resulted in the following –
- Aphasias Broca’s and Wernicke’s
- Auditory verbal agnosia
- Auditory agnosia
- Apraxia of Speech
- Visuospatial Dysgnosia
Stroke – Not not all these symptoms apply to me
- Contralateral loss of pain and temperature sensations.
- Visual field defects (contralateral hemianopia with macular sparing).
- Prosopagnosia with bilateral obstruction of the lingual and fusiform gyri.
- Superior Alternating Syndrome (Weber’s syndrome)
- Contralateral deficits of facial nerve (only lower face, upper face receives bilateral input), vagus nerve and hypoglossal nerve
- Ipsilateral deficit of oculomotor nerve
- Horner’s Syndrome
Peripheral territory (Cortical branches) – Not not all these symptoms apply to me
- Homonymous hemianopia (often upper quadrantic): Calcarine cortex or optic radiation nearby.
- Bilateral homonymous hemianopia, cortical blindness, awareness or denial of blindness; tactile naming, achromatopia (color blindness), failure to see to-and-fro movements, inability to perceive objects not centrally located, apraxia of ocular movements, inability to count or enumerate objects, tendency to run into things that the patient sees and tries to avoid: Bilateral occipital lobe with possibly the parietal lobe involved.
- Verbal dyslexia without agraphia, color anomia: Dominant calcarine lesion and posterior part of corpus callosum.
- Memory defect: Hippocampal lesion bilaterally or on the dominant side only.
- Topographic disorientation and prosopagnosia: Usually with lesions of nondominant, calcarine, and lingual gyrus.
- Simultanagnosia, hemivisual neglect: Dominant visual cortex, contralateral hemisphere.
- Unformed visual hallucinations, peduncular hallucinosis, metamorphopsia, teleopsia, illusory visual spread, palinopsia, distortion of outlines, central photophobia: Calcarine cortex.
- Complex hallucinations: Usually nondominant hemisphere.
Due to a silent stroke within the womb which is to do with
- Visual field deficits
- Pain Agnosia (Analgesia)
Additional injury to the left hemisphere to the such as
- Semantic Agnosia
- Visual agnosias
- Visual Memory
- Gertsmann Syndrome (reading and writing difficulties, left right confusion, Finger agnosia)
It is important to know the trajectory and the why’s and the hows? of how someone has Autism, all this being said I will not let this DEFINE my PERSONHOOD but it does continue to explain why a process and communicate in a particular way. 🙂
Paul Isaacs 2014